Functional model · SBNFA™

Neurosensory deprogramming

Neurosensory deprogramming refers, in the functional model developed by Blue Portance from the work of Pierre Ivaldi, to the progressive loss of the nervous system’s ability to calibrate its response correctly based on the sensory information it actually receives.

Scientific basis

The model draws on phenomena documented separately by research: sensitization of the nervous system, neuroplasticity, learning and sensory integration. It does not correspond to an IASP category or to a medical diagnosis; it places these mechanisms in perspective to describe a functional evolution.

Blue Portance / SBNFA™ reading

In the Blue Portance reading, the nervous system’s response (thresholds, intensity, spread of pain) can gradually drift away from the sensory information actually received. This drift is not a mandatory stage after hypersensitization, and it does not imply an erasure of neural circuits. The model serves to organize the analysis; it is not a universal explanation of chronic pain.

Contribution to the analysis of sitting

Seating analysis does not assess neurosensory deprogramming. It describes support conditions and their variability, that is, part of the sensory information the nervous system receives in sitting.

Not to be confused with

Central sensitization, a neurophysiological mechanism defined by the IASP; nervous system hypersensitization, the Blue Portance reading of a state of heightened reactivity, which is not defined as a loss of calibration; nociplastic pain, an IASP pain category; neurosensory reprogramming, gradual relearning, which is not the exact reverse of deprogramming.

Sources and scope

Interpretive functional model by Blue Portance, derived from the work of Pierre Ivaldi. It is not a diagnosis, a recognized medical mechanism or a pain category.